A case report of epilepsia partialis continua as a rare initial presentation of diabetic ketoacidosis.
Keywords:Hyperglycemia, magnetic resonance imaging, seizures, Faciobrachial myoclonus, Epilepsia Partialis Continua, diabetic ketoacidosis
The horizon of movement disorders in diabetes mellitus is expanding. While chorea and ballism and rarely different types of myoclonus are well-recognised movement disorders as presenting manifestation of non-ketotic hyperglycemic states, it is not well documented in DKA. Here we report a 53 year old female presenting with Faciobrachial myoclonic seizures as an initial manifestation of diabetic ketoacidosis and treatment of seizures with adequate glycemic control and correction of DKA
Maccario M, Messis CP, Vastola F. Focal seizures as a manifestation of hyperglycaemia without ketoacidosis. Neurology 1965;15:195-206.
Stoner G: Hyperosmolar hyperglycemic state. Am Fam Physician. 2017, 96:729-736.
Hennis A, Corbin D, Fraser H. Focal seizures and non-ketotic hyperglycaemia. J Neurol Neurosurg Psychiatry. 1992;55:195–7.
Roberts E, Rothstein M, Baxter CF. Some metabolic studies of gamma- aminobutyric acid. Proc Soc Exp Biol Med. 1958;97:796–802.
Guisado R, Arieff AI. Neurologic manifestations of diabetic comas: Correlation with biochemical alterations in the brain. Metabolism. 1975;24:665–79.
Duckrow RB. Decreased cerebral blood flow during acute hyperglycemia. Brain Res. 1995;703:145–50.
Bhagwat NM, Joshi AS, Rao G, Varthakavi PK. Uncontrolled hyperglycaemia: A reversible cause of hemichorea-hemiballismus. BMJ
Case Rep 2013. 2013 pii : bcr2013010229.
Wintermark M, Fischbein NJ, Mukherjee P, Yuh EL, Dillon WP. Unilateral putaminal CT, MR, and diffusion abnormalities secondary to nonketotic hyperglycemia in the setting of acute neurologic symptoms mimicking stroke. AJNR Am J Neuroradiol. 2004;25:975–6
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